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Conclusions Changes in intra-portal insulin levels play an intricate yet permissive role in the regulation of the GH/IGF-I axis by either stimulating or suppressing hepatic GH sensitivity and hepatic IGF-I generation, probably via an insulin-mediated regulation of the hepatic GHR expression

case of progressive multifocal leukoencephalopathy under dimethyl fumarate treatment without severe lymphopenia or immunosenescence

Abstract Background Alzheimers disease (AD) is the leading cause of dementia, characterized by a complex pathogenesis that complicates the development of effective treatments

Inactivation-reactivation of aconitase in Escherichia coli